What is HDL?
- High-Density Lipo-proteins (HDL) are the “good cholesterol”
- HDL removes low-density lipoproteins (LDL- the “bad cholesterol”) and macrophages from blood and artery walls
- Recent advances in both animal studies and clinical trials indicate that increasing HDL levels could result in additional benefits to those achieved by reducing LDL levels using statins
- HDL has also demonstrated anti-adhesive properties, preventing the colonization of pathogens like E. coli and Salmonella typhimurium
- These HDL naturally occur in egg yolk
- Ecovatec’s proprietary processes remove most of the LDL
Study shows Egg Yolk HDL effective against Cardiovascular Disease
These findings indicate that treatment with egg yolk HDL increased serum HDLC and decreased atherosclerotic plaque size in rabbits. Thus, egg yolk HDL can be considered as an antiatherosclerosis agent in treatment of patients with cardiovascular diseases.1
Eftekhar et. al., “The Prevention and Treatment Effects of Egg Yolk High Density Lipoprotein on the Formation of Atherosclerosis Plaque in Rabbits,” Iranian J Basic Med Sci. 2015 Apr; 18(4):343-349. Link to Study
- The primary function of HDL is to remove excess cholesterol and carry the excess LDL to the liver to be metabolized into bile salt
- Stimulates natural HDL production
- Promotes endothelial generation of Nitric Oxide (invitro) and improves endothelial function (anti-inflammation in arteries)
- Antagonizing platelet activity and inactivating coagulation cascade
- Nutraceutical additive for heart health products
- Functional foods
- Sports Nutrition
Images below are taken from the American Heart Association (http://www.heart.org)
Cholesterol, LDL and HDL
Cholesterol isn’t just something that sits in your body like fat around your waist. It’s carried through your bloodstream by carriers made of fat (lipid) and proteins. These are called – no big surprise – lipoproteins.
Two types of lipoproteins carry cholesterol to and from cells. One is low-density lipoprotein, or LDL. The other is high-density lipoprotein, or HDL.
The amount of each type of cholesterol in your blood can be measured by a blood test.
Low Density Lipoprotein (LDL)
LDL = Bad Cholesterol
LDL cholesterol is called “bad” cholesterol.
Think of it as less desirable or even lousy cholesterol, because it contributes to fatty buildups in arteries (atherosclerosis).
High Density Lipoprotein (HDL)
HDL = Good Cholesterol
HDL cholesterol is “good” cholesterol. Think of it as the “healthy” cholesterol, so higher levels are better. Experts believe HDL acts as a scavenger, carrying LDL cholesterol away from the arteries and back to the liver. There it’s broken down and passed from the body.
A healthy HDL cholesterol level may protect against heart attack and stroke. Studies show low levels of HDL cholesterol increase the risk of heart disease. HDL cholesterol does not completely eliminate LDL cholesterol. Only one-fourth to one-third of blood cholesterol is carried by HDL.
Total Cholesterol Score
Total Cholesterol Score
Coronary artery disease (CAD) occurs when the inside (the lumen) of one or more coronary arteries narrows, limiting the flow of oxygen-rich blood to surrounding heart muscle tissue.
Atherosclerosis is the process that causes the artery wall to get thick and stiff. It can lead to complete blockage of the artery, which can cause a heart attack.
The disease process begins when LDL (“bad” cholesterol) deposits cholesterol in the artery wall. The body has an immune response to protect itself and sends white blood cells called macrophages to engulf the invading cholesterol in the artery wall.
When the macrophages are full of cholesterol, they are called foam cells because of their appearance.
As more foam cells collect in the artery wall, a fatty streak develops between the intima and the media. If the process is not stopped, the fatty streak becomes a plaque, which pushes the intima into the lumen, narrowing the blood flow.
The plaque develops a fibrous coating on its outer edges. But if cholesterol continues to collect in foam cells inside the plaque, the fibrous outer coating can weaken and eventually rupture.
Smaller arteries downstream from the rupture can quickly become blocked. Over time, a clot may develop at the rupture site and completely block the artery.